Wednesday, 25 November 2009 14:06

Family Explanations of Anorexia

Written by Keiron Walsh
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Family Environment theories of Anorexia cite dysfunctional family interaction as the cause of eating disorders. According to a number of researchers (e.g., Strober, 1992; Irving, 1990), as many as half of the families of people with eating disorders have a long history of emphasising thinness, physical appearance and dieting.

Family systems theorists, such as Lundholm & Waters (1991), claim that Eating disorders, such as anorexia are a sign of a much larger family pathology. Minuchin et al (1978) claims that it is a particular type of dysfunctional family, an enmeshed family, that leads to eating disorders.

Enmeshed family members over involved with each others lives, have no clear sense of autonomy and excessive togetherness leads members to speak for each other and intrude on each other’s thoughts & feelings. Parents in enmeshed families do not see their children as individuals in their own right, but as appendages which make their lives more complete. In reality, the closeness of an enmeshed family is an illusion.

Minuchin and his colleagues (1978) explain the development of eating disorders as following a particular pattern: as the adolescent child pushes for independence, it threatens the family illusion of togetherness. The child responds by taking on a “sick role” (developing an eating disorder) which means that the family “rallies round”. This “rallying round” restores the illusion of togetherness. Minuchin also uses this explanation to account for other disorders, such as psychosomatic illnesses.

Evaluation of Family Enmeshment as a Cause of Anorexia

Arthur Crisp and his colleagues (1974) tested the idea that the families of people with eating disorders would become unstable if their child recovered. He found that when the children gained weight the parents did show a significant increase in depression and anxiety. This supports the theory because it suggests that the family is stressed by the threat of not having a crisis to bring them together. However, it is possible that the increase in anxiety and depression may be in response to the stresses of weight restoration therapy (Strober, 1992).

There is more evidence, however, that family dysfunction, is associated more with bulimia nervosa than with anorexia (Fornari et al, 1999). Moreover, although family enmeshment predicts psychological disorder in general, the following family climate variables seem to be more related to eating disorders:

  • Family Body Satisfaction
  • Family Social Appearance Orientation
  • Family Achievement Emphasis

An issue with evidence in support of family theories is that it is very difficult to carry out research that is able to separate effects that are due to family interactions and those that are due to genetic influences. It could be that genes that lead to obsessive behaviour, for example, contribute to both anorexia and family dysfunction.

Last modified on Saturday, 17 April 2010 15:52

1 Comment

  • Comment Link Gulsanga Thursday, 16 June 2011 10:29 posted by Gulsanga

    Is this a psychodynamic explanation?

    There is one psychodynamic explanation which is quite similar. in Freudian terms eating and sex are seen as being linked to one another. The denial of eating symbolises the denial to sexual maturity; usually anorexics go back to an appearance of pre-puberty age ie breasts stop deveolping and menstruation stops.

    freud believed that this is because the teenager uncounsiously does not want to grow up, hence, they develop this disorder so that they get attention and become more dependent on their parents as they did as children.

    However, these explantions are overlooking other age groups, and sees the disorder just as a strategy that teenagers use to cope with the changes that are happening to them. there are some teenagers/people who hide the fact that they arent eating properly from their parents, thus, these explanations are reductionist as they dont explain all cases of anorexics

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Keiron Walsh

Keiron Walsh

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